A young, massive planet is orbiting in an unusual place in its star system, and it’s leading researchers to revive a long-debated view of how giant planets can form.

The protoplanet, nine times the mass of Jupiter, is too far away from its star to have formed by accreting matter piece by piece, images suggest. Instead, the massive world probably formed all at once in a violent implosion of gas and dust, researchers report April 4 in Nature Astronomy.

“My first reaction was, there’s no way this can be true,” says Thayne Currie, an astrophysicist at the Subaru Telescope headquartered in Hilo, Hawaii.

For years, astronomers have debated the ways in which giant planets might form (SN: 12/3/10). In the “core accretion” story, a planet starts out as small bits of matter within a disk of gas, dust and ice swirling around a young star. The clumps continue to accrete other matter, growing to become the core of the planet. Out past a certain distance from the star, that core then accumulates a thick blanket of hydrogen and helium, turning it into a bloated, gassy world.

But the new planet, orbiting a star called AB Aurigae, is in the outskirts of its system, where there’s less matter to gather into a core. In this position, the core can’t become massive enough to create its gaseous envelope. The planet’s remote location, Currie and colleagues argue, makes it more likely to form via “disk instability,” where the disk around the star breaks into planet-sized fragments. The fragments then rapidly collapse in on themselves, drawn together by their own gravity, and clump together, forming a giant planet.
Using the Subaru Telescope atop Mauna Kea, Currie and colleagues observed AB Aurigae periodically from 2016 to 2020. NASA’s Hubble Space Telescope also observed the star repeatedly over 13 years. Looking at all these images, the team saw a bright spot next to the star. The bright clump was a clear protoplanet, named AB Aur b, orbiting nearly 14 billion kilometers from its star — roughly 3 times as far as Neptune is from the sun.

In the images, AB Aur b looked like it was straight out of a simulation of planet formation by disk instability, Currie says. Except it was real.
“For the longest time, I never believed that planet formation by disk instability could actually work,” he says.

Because AB Aur b is still growing, embedded in the young star’s disk, it could help to explain how the handful of known massive planets orbiting far from their stars formed.

“We only know maybe a few dozen total of these types of planets,” says Quinn Konopacky, an astrophysicist at the University of California, San Diego who was not involved in the research. “Every single one that we find is basically precious.”

It’s difficult to distinguish whether a planet formed by core accretion or disk instability through observations alone, Konopacky says. The fact that AB Aur b is at such a wide separation from its star is “good evidence” that disk instability is what’s happening, she says. Still, “I think that there’s a lot more work to be done and other ways that we can try to assess if that’s what’s going on in the system.”

Both Konopacky and Currie say this research represents only the second direct observation of a protoplanet (SN: 7/2/18). Oftentimes, researchers have trouble distinguishing an actual forming planet from a planetary disk.

The recently launched James Webb Space Telescope could help researchers understand these anomalous gas giants very distant from their stars by studying the AB Aurigae system and others like it, Currie says (SN: 1/24/22). “I think this will spur a lot of debates and follow-up studies by other researchers.”

Deep in the human brain, a very specific kind of cell dies during Parkinson’s disease.

For the first time, researchers have sorted large numbers of human brain cells in the substantia nigra into 10 distinct types. Just one is especially vulnerable in Parkinson’s disease, the team reports May 5 in Nature Neuroscience. The result could lead to a clearer view of how Parkinson’s takes hold, and perhaps even ways to stop it.

The new research “goes right to the core of the matter,” says neuroscientist Raj Awatramani of Northwestern University Feinberg School of Medicine in Chicago. Pinpointing the brain cells that seem to be especially susceptible to the devastating disease is “the strength of this paper,” says Awatramani, who was not involved in the study.

Parkinson’s disease steals people’s ability to move smoothly, leaving balance problems, tremors and rigidity. In the United States, nearly 1 million people are estimated to have Parkinson’s. Scientists have known for decades that these symptoms come with the death of nerve cells in the substantia nigra. Neurons there churn out dopamine, a chemical signal involved in movement, among other jobs (SN: 9/7/17).

But those dopamine-making neurons are not all equally vulnerable in Parkinson’s, it turns out.

“This seemed like an opportunity to … really clarify which kinds of cells are actually dying in Parkinson’s disease,” says Evan Macosko, a psychiatrist and neuroscientist at Massachusetts General Hospital in Boston and the Broad Institute of MIT and Harvard.
The tricky part was that dopamine-making neurons in the substantia nigra are rare. In samples of postmortem brains, “we couldn’t survey enough of [the cells] to really get an answer,” Macosko says. But Abdulraouf Abdulraouf, a researcher in Macosko’s laboratory, led experiments that sorted these cells, figuring out a way to selectively pull the cells’ nuclei out from the rest of the cells present in the substantia nigra. That enrichment ultimately led to an abundance of nuclei to analyze.

By studying over 15,000 nuclei from the brains of eight formerly healthy people, the researchers further sorted dopamine-making cells in the substantia nigra into 10 distinct groups. Each of these cell groups was defined by a specific brain location and certain combinations of genes that were active.

When the researchers looked at substantia nigra neurons in the brains of people who died with either Parkinson’s disease or the related Lewy body dementia, the team noticed something curious: One of these 10 cell types was drastically diminished.

These missing neurons were identified by their location in the lower part of the substantia nigra and an active AGTR1 gene, lab member Tushar Kamath and colleagues found. That gene was thought to serve simply as a good way to identify these cells, Macosko says; researchers don’t know whether the gene has a role in these dopamine-making cells’ fate in people.

The new finding points to ways to perhaps counter the debilitating diseases. Scientists have been keen to replace the missing dopamine-making neurons in the brains of people with Parkinson’s. The new study shows what those cells would need to look like, Awatramani says. “If a particular subtype is more vulnerable in Parkinson’s disease, maybe that’s the one we should be trying to replace,” he says.

In fact, Macosko says that stem cell scientists have already been in contact, eager to make these specific cells. “We hope this is a guidepost,” Macosko says.

The new study involved only a small number of human brains. Going forward, Macosko and his colleagues hope to study more brains, and more parts of those brains. “We were able to get some pretty interesting insights with a relatively small number of people,” he says. “When we get to larger numbers of people with other kinds of diseases, I think we’re going to learn a lot.”

An act of acrobatics keeps males of one orb-weaving spider species from becoming their mates’ post-sex snack.

After mating, Philoponella prominens males catapult away from females at speeds up to nearly 90 centimeters per second, researchers report April 25 in Current Biology. Other spiders jump to capture prey or avoid predators (SN: 3/16/19). But P. prominens is unique among spiders in that males soar through the air to avoid sexual cannibalism, the researchers say.

P. prominens is a social species that’s native to countries such as Japan and Korea. Up to 300 individual spiders can come together to weave an entire neighborhood of webs. While studying P. prominens’ sexual behavior, arachnologist Shichang Zhang and colleagues noticed that sex seemed to always end with a catapulting male. But the movement was “so fast that common cameras could not record the details,” says Zhang, of Hubei University in Wuhan, China.

High-resolution video of mating partners clocked the male arachnids’ speed from around 32 cm/s to 88 cm/s, the researchers report. That’s equal to just under 1 mile per hour to nearly 2 mph.
The jump looks a little like the start of a backstroke swimming race, Zhang says. Males hold the tips of their front legs against a female’s body. The spiders then use hydraulic pressure to extend a joint in those legs, quickly launching a male off a female before she can capture and eat him.

Of 155 successful mating rituals that the researchers observed, 152 males catapulted to survival. The remaining three that didn’t fell victim to their partner. Female spiders also ate all 30 males that the team stopped from jumping to freedom with a paintbrush.

These male orb weavers probably acquired their jumping abilities to counter females’ cannibalistic tendencies, Zhang says. The spiders’ leap to survival is a “fantastic kinetic performance.”